N-아세틸시스테인(NAC) 그림

43 동료 심사 연구의 그림

전체 생강 아연 이눌린 커큐민 락타아제 베르베린 차전자피 비피도박테리움 롱검 비피도박테리움 락티스 비피도박테리움 비피덤 사카로마이세스 보울라디 락토바실루스 가세리 락토바실루스 플란타룸 락토바실루스 애시도필루스 부티레이트(나트륨/칼슘 부티레이트) 페퍼민트 오일 바실루스 코아굴란스 췌장효소(판크렐리파아제) 알로에 베라(내엽 젤) 알파-갈락토시다아제 소 초유 오메가-3 지방산(EPA/DHA) 비타민 A 비타민 D 프럭토올리고당(FOS) 갈락토올리고당(GOS) L-글루타민 중쇄중성지방(MCT 오일) N-아세틸시스테인(NAC)

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Figure 11

Figure 7 Histological examination of the safety of selenium-containing amino acids. Lung, kidney, heart, liver, and spleen tissues were collected, H&E stained, and analyzed for the safety profile of selenium-containing …

Selenium-Containing Amino Acids Protect Dextran Sulfate Sodium-Induced Colitis via Ameliorating Oxidative Stress …

Figure 6 Micrograph

Immunofluorescence imaging of Ku70/Ku80 localization in gastric epithelial cells, showing nuclear protein retention in alpha-lipoic acid-treated cells despite H. pylori infection.

α-Lipoic Acid Inhibits Apoptosis by Suppressing the Loss of Ku Proteins in …

Figure 7 Chart

Quantification of DNA double-strand break repair efficiency in H. pylori-infected gastric cells, indicating that alpha-lipoic acid supports the Ku-dependent non-homologous end joining pathway.

α-Lipoic Acid Inhibits Apoptosis by Suppressing the Loss of Ku Proteins in …

Figure 8 Chart

Cell viability analysis across different alpha-lipoic acid concentrations in H. pylori-infected gastric epithelial cells, establishing the effective dose range for cytoprotection.

α-Lipoic Acid Inhibits Apoptosis by Suppressing the Loss of Ku Proteins in …

Figure 9 Diagram

Signaling pathway analysis showing the molecular mechanism by which alpha-lipoic acid preserves Ku protein levels and inhibits apoptosis in H. pylori-infected gastric cells.

α-Lipoic Acid Inhibits Apoptosis by Suppressing the Loss of Ku Proteins in …

Figure 2 Micrograph

Liver histopathology from colitis-induced mice with and without oral NAC supplementation, evaluating extraintestinal hepatic manifestations including steatosis and inflammatory infiltration.

Extraintestinal Manifestations in Induced Colitis: Controversial Effects of N-Acetylcysteine on Colon, Liver, …

Figure 3 Micrograph

Kidney tissue histology showing renal changes associated with induced colitis, and the effects of N-acetylcysteine on renal inflammation and oxidative damage markers.

Extraintestinal Manifestations in Induced Colitis: Controversial Effects of N-Acetylcysteine on Colon, Liver, …

Figure 4 Chart

Oxidative stress biomarkers measured in colon, liver, and kidney tissues, comparing malondialdehyde (MDA) and glutathione (GSH) levels across treatment groups.

Extraintestinal Manifestations in Induced Colitis: Controversial Effects of N-Acetylcysteine on Colon, Liver, …

Figure 5 Chart

Inflammatory cytokine levels (such as TNF-alpha, IL-6, or IL-1beta) in colonic and extraintestinal tissues, assessing whether NAC modulates systemic inflammation originating from the gut.

Extraintestinal Manifestations in Induced Colitis: Controversial Effects of N-Acetylcysteine on Colon, Liver, …

Figure 6 Chart

Colon length measurements and macroscopic damage scoring in the colitis model, providing gross anatomical evidence of disease severity and NAC treatment response.

Extraintestinal Manifestations in Induced Colitis: Controversial Effects of N-Acetylcysteine on Colon, Liver, …

Figure 7 Chart

Summary of controversial findings regarding NAC's differential effects across organs, with protective trends observed in some tissues but potential adverse signals in others.

Extraintestinal Manifestations in Induced Colitis: Controversial Effects of N-Acetylcysteine on Colon, Liver, …

Figure 2

Scheme 1.Flowchart 1:The coding for mucopenetrative liposomal formulation compositions (MP1-MP6).The coding for mucopenetrative liposomal formulation compositions (MP1-MP6).