描述
Immune modulation by probiotic-fermented herbal bioactives in the context of metabolic inflammation. Toll-like receptor signaling, NF-kB pathway regulation, and cytokine balance are presented as mechanistic targets.
Figure 10
DiagramSource Paper
Gut Microbiota-Targeted Therapeutics for Metabolic Disorders: Mechanistic Insights into the Synergy of Probiotic-Fermented Herbal Bioactives.Cite This Figure
![Figure 10: Immune modulation by probiotic-fermented herbal bioactives in the context of metabolic inflammation. Toll-like receptor signaling, NF-kB pathway regulation, and cytokine balance are presented as mechanistic targets.]() > Source: Yue Fan et al. "Gut Microbiota-Targeted Therapeutics for Metabolic Disorders: Mechanistic Insigh." *International journal of molecular sciences*, 2025. PMID: [40564947](https://pubmed.ncbi.nlm.nih.gov/40564947/)
<figure> <img src="" alt="Immune modulation by probiotic-fermented herbal bioactives in the context of metabolic inflammation. Toll-like receptor signaling, NF-kB pathway regulation, and cytokine balance are presented as mechanistic targets." /> <figcaption>Figure 10. Immune modulation by probiotic-fermented herbal bioactives in the context of metabolic inflammation. Toll-like receptor signaling, NF-kB pathway regulation, and cytokine balance are presented as mechanistic targets.<br> Source: Yue Fan et al. "Gut Microbiota-Targeted Therapeutics for Metabolic Disorders: Mechanistic Insigh." <em>International journal of molecular sciences</em>, 2025. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/40564947/">40564947</a></figcaption> </figure>