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In vivo intestinal inflammation modulated by AIEC capacity to metabolize ethanolamine and fucose/rhamnose in an IBD-susceptible mouse model. Metabolite utilization genes influence colonization and inflammatory outcomes.

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![Figure 7: In vivo intestinal inflammation modulated by AIEC capacity to metabolize ethanolamine and fucose/rhamnose in an IBD-susceptible mouse model. Metabolite utilization genes influence colonization and inflammatory outcomes.]()

> Source: Shiying Zhang et al. "Mucosal metabolites fuel the growth and virulence of E. coli linked to Crohn's d." *JCI insight*, 2022. PMID: [35413017](https://pubmed.ncbi.nlm.nih.gov/35413017/)
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  <img src="" alt="In vivo intestinal inflammation modulated by AIEC capacity to metabolize ethanolamine and fucose/rhamnose in an IBD-susceptible mouse model. Metabolite utilization genes influence colonization and inflammatory outcomes." />
  <figcaption>Figure 7. In vivo intestinal inflammation modulated by AIEC capacity to metabolize ethanolamine and fucose/rhamnose in an IBD-susceptible mouse model. Metabolite utilization genes influence colonization and inflammatory outcomes.<br>  Source: Shiying Zhang et al. "Mucosal metabolites fuel the growth and virulence of E. coli linked to Crohn's d." <em>JCI insight</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/35413017/">35413017</a></figcaption>
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