描述
Inflammatory cytokine expression levels in colonic tissue from BPTES-treated versus untreated IL-10-knockout mice. Pro-inflammatory markers including TNF-alpha and IL-17 are quantified by qPCR.
Figure 2
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Spontaneous colitis in IL-10-deficient mice was ameliorated via inhibiting glutaminase1.Cite This Figure
![Figure 2: Inflammatory cytokine expression levels in colonic tissue from BPTES-treated versus untreated IL-10-knockout mice. Pro-inflammatory markers including TNF-alpha and IL-17 are quantified by qPCR.]() > Source: Jing Li et al. "Spontaneous colitis in IL-10-deficient mice was ameliorated via inhibiting gluta." *Journal of cellular and molecular medicine*, 2019. PMID: [31211512](https://pubmed.ncbi.nlm.nih.gov/31211512/)
<figure> <img src="" alt="Inflammatory cytokine expression levels in colonic tissue from BPTES-treated versus untreated IL-10-knockout mice. Pro-inflammatory markers including TNF-alpha and IL-17 are quantified by qPCR." /> <figcaption>Figure 2. Inflammatory cytokine expression levels in colonic tissue from BPTES-treated versus untreated IL-10-knockout mice. Pro-inflammatory markers including TNF-alpha and IL-17 are quantified by qPCR.<br> Source: Jing Li et al. "Spontaneous colitis in IL-10-deficient mice was ameliorated via inhibiting gluta." <em>Journal of cellular and molecular medicine</em>, 2019. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/31211512/">31211512</a></figcaption> </figure>