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Extended analysis of the host-microbiome interaction in mitochondria-deficient mice reveals additional pathways linking epithelial energy metabolism to mucosal immune responses.

Figure 10

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Source Paper

Interplay of gut microbiota and host epithelial mitochondrial dysfunction is necessary for the development of spontaneous intestinal inflammation in mice.

Microbiome (2023)

PMID: 37978573

DOI: 10.1186/s40168-023-01686-9

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![Figure 10: Extended analysis of the host-microbiome interaction in mitochondria-deficient mice reveals additional pathways linking epithelial energy metabolism to mucosal immune responses.]()

> Source: Kibrom M Alula et al. "Interplay of gut microbiota and host epithelial mitochondrial dysfunction is nec." *Microbiome*, 2023. PMID: [37978573](https://pubmed.ncbi.nlm.nih.gov/37978573/)

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  <img src="" alt="Extended analysis of the host-microbiome interaction in mitochondria-deficient mice reveals additional pathways linking epithelial energy metabolism to mucosal immune responses." />
  <figcaption>Figure 10. Extended analysis of the host-microbiome interaction in mitochondria-deficient mice reveals additional pathways linking epithelial energy metabolism to mucosal immune responses.<br>  Source: Kibrom M Alula et al. "Interplay of gut microbiota and host epithelial mitochondrial dysfunction is nec." <em>Microbiome</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/37978573/">37978573</a></figcaption>
</figure>

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Figure 10

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