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Immune cell infiltration analysis in the intestinal lamina propria compares inflammatory cell populations between mitochondria-deficient and wild-type mice.

Figure 7

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Source Paper

Interplay of gut microbiota and host epithelial mitochondrial dysfunction is necessary for the development of spontaneous intestinal inflammation in mice.

Microbiome (2023)

PMID: 37978573

DOI: 10.1186/s40168-023-01686-9

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![Figure 7: Immune cell infiltration analysis in the intestinal lamina propria compares inflammatory cell populations between mitochondria-deficient and wild-type mice.]()

> Source: Kibrom M Alula et al. "Interplay of gut microbiota and host epithelial mitochondrial dysfunction is nec." *Microbiome*, 2023. PMID: [37978573](https://pubmed.ncbi.nlm.nih.gov/37978573/)

<figure>
  <img src="" alt="Immune cell infiltration analysis in the intestinal lamina propria compares inflammatory cell populations between mitochondria-deficient and wild-type mice." />
  <figcaption>Figure 7. Immune cell infiltration analysis in the intestinal lamina propria compares inflammatory cell populations between mitochondria-deficient and wild-type mice.<br>  Source: Kibrom M Alula et al. "Interplay of gut microbiota and host epithelial mitochondrial dysfunction is nec." <em>Microbiome</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/37978573/">37978573</a></figcaption>
</figure>

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Figure 7

Source Paper

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