설명
Comparative cell imaging showing gastric epithelial cells under H. pylori infection with and without N-acetylcysteine (NAC) treatment, demonstrating NAC's protective effects against oxidative DNA damage.
Figure 5
MicrographSource Paper
N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway Activation Induced by Helicobacter pylori Infection.Cite This Figure
![Figure 5: Comparative cell imaging showing gastric epithelial cells under H. pylori infection with and without N-acetylcysteine (NAC) treatment, demonstrating NAC's protective effects against oxidative DNA damage.]() > Source: Chuan Xie et al. "N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway ." *Oxidative medicine and cellular longevity*, 2018. PMID: [29854076](https://pubmed.ncbi.nlm.nih.gov/29854076/)
<figure> <img src="" alt="Comparative cell imaging showing gastric epithelial cells under H. pylori infection with and without N-acetylcysteine (NAC) treatment, demonstrating NAC's protective effects against oxidative DNA damage." /> <figcaption>Figure 5. Comparative cell imaging showing gastric epithelial cells under H. pylori infection with and without N-acetylcysteine (NAC) treatment, demonstrating NAC's protective effects against oxidative DNA damage.<br> Source: Chuan Xie et al. "N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway ." <em>Oxidative medicine and cellular longevity</em>, 2018. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/29854076/">29854076</a></figcaption> </figure>