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In vivo intestinal inflammation modulated by AIEC capacity to metabolize ethanolamine and fucose/rhamnose in an IBD-susceptible mouse model. Metabolite utilization genes influence colonization and inflammatory outcomes.
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Mucosal metabolites fuel the growth and virulence of E. coli linked to Crohn's disease.Cite This Figure
![Figure 7: In vivo intestinal inflammation modulated by AIEC capacity to metabolize ethanolamine and fucose/rhamnose in an IBD-susceptible mouse model. Metabolite utilization genes influence colonization and inflammatory outcomes.]() > Source: Shiying Zhang et al. "Mucosal metabolites fuel the growth and virulence of E. coli linked to Crohn's d." *JCI insight*, 2022. PMID: [35413017](https://pubmed.ncbi.nlm.nih.gov/35413017/)
<figure> <img src="" alt="In vivo intestinal inflammation modulated by AIEC capacity to metabolize ethanolamine and fucose/rhamnose in an IBD-susceptible mouse model. Metabolite utilization genes influence colonization and inflammatory outcomes." /> <figcaption>Figure 7. In vivo intestinal inflammation modulated by AIEC capacity to metabolize ethanolamine and fucose/rhamnose in an IBD-susceptible mouse model. Metabolite utilization genes influence colonization and inflammatory outcomes.<br> Source: Shiying Zhang et al. "Mucosal metabolites fuel the growth and virulence of E. coli linked to Crohn's d." <em>JCI insight</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/35413017/">35413017</a></figcaption> </figure>