説明
Extended analysis of the host-microbiome interaction in mitochondria-deficient mice reveals additional pathways linking epithelial energy metabolism to mucosal immune responses.
Figure 10
ChartSource Paper
Interplay of gut microbiota and host epithelial mitochondrial dysfunction is necessary for the development of spontaneous intestinal inflammation in mice.Cite This Figure
![Figure 10: Extended analysis of the host-microbiome interaction in mitochondria-deficient mice reveals additional pathways linking epithelial energy metabolism to mucosal immune responses.]() > Source: Kibrom M Alula et al. "Interplay of gut microbiota and host epithelial mitochondrial dysfunction is nec." *Microbiome*, 2023. PMID: [37978573](https://pubmed.ncbi.nlm.nih.gov/37978573/)
<figure> <img src="" alt="Extended analysis of the host-microbiome interaction in mitochondria-deficient mice reveals additional pathways linking epithelial energy metabolism to mucosal immune responses." /> <figcaption>Figure 10. Extended analysis of the host-microbiome interaction in mitochondria-deficient mice reveals additional pathways linking epithelial energy metabolism to mucosal immune responses.<br> Source: Kibrom M Alula et al. "Interplay of gut microbiota and host epithelial mitochondrial dysfunction is nec." <em>Microbiome</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/37978573/">37978573</a></figcaption> </figure>