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Mechanisms linking gut microbiota dysbiosis to atherosclerosis progression, including TMAO production, bile acid metabolism, and systemic inflammation pathways. The data from this meta-analysis of animal studies support a role for microbiome modulation in cardiovascular protection.
Figure 6
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Effects of Berries, Phytochemicals, and Probiotics on Atherosclerosis through Gut Microbiota Modification: A Meta-Analysis of Animal Studies.Cite This Figure
![Figure 6: Mechanisms linking gut microbiota dysbiosis to atherosclerosis progression, including TMAO production, bile acid metabolism, and systemic inflammation pathways. The data from this meta-analysis of animal studies support a role for microbiome modulation in cardiovascular protection.]() > Source: Leila Khalili et al. "Effects of Berries, Phytochemicals, and Probiotics on Atherosclerosis through Gu." *International journal of molecular sciences*, 2023. PMID: [36834497](https://pubmed.ncbi.nlm.nih.gov/36834497/)
<figure> <img src="" alt="Mechanisms linking gut microbiota dysbiosis to atherosclerosis progression, including TMAO production, bile acid metabolism, and systemic inflammation pathways. The data from this meta-analysis of animal studies support a role for microbiome modulation in cardiovascular protection." /> <figcaption>Figure 6. Mechanisms linking gut microbiota dysbiosis to atherosclerosis progression, including TMAO production, bile acid metabolism, and systemic inflammation pathways. The data from this meta-analysis of animal studies support a role for microbiome modulation in cardiovascular protection.<br> Source: Leila Khalili et al. "Effects of Berries, Phytochemicals, and Probiotics on Atherosclerosis through Gu." <em>International journal of molecular sciences</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/36834497/">36834497</a></figcaption> </figure>