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Dysbiosis in atopic dermatitis is linked to skin immune responses, illustrating how altered gut and skin microbial communities contribute to eczema pathogenesis. Bifidobacterial depletion correlates with increased Th2-skewed inflammation.

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![Figure 7: Dysbiosis in atopic dermatitis is linked to skin immune responses, illustrating how altered gut and skin microbial communities contribute to eczema pathogenesis. Bifidobacterial depletion correlates with increased Th2-skewed inflammation.]()

> Source: Luisa Marras et al. "The Role of Bifidobacteria in Predictive and Preventive Medicine: A Focus on Ecz." *Microorganisms*, 2021. PMID: [33919907](https://pubmed.ncbi.nlm.nih.gov/33919907/)
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  <img src="" alt="Dysbiosis in atopic dermatitis is linked to skin immune responses, illustrating how altered gut and skin microbial communities contribute to eczema pathogenesis. Bifidobacterial depletion correlates with increased Th2-skewed inflammation." />
  <figcaption>Figure 7. Dysbiosis in atopic dermatitis is linked to skin immune responses, illustrating how altered gut and skin microbial communities contribute to eczema pathogenesis. Bifidobacterial depletion correlates with increased Th2-skewed inflammation.<br>  Source: Luisa Marras et al. "The Role of Bifidobacteria in Predictive and Preventive Medicine: A Focus on Ecz." <em>Microorganisms</em>, 2021. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/33919907/">33919907</a></figcaption>
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