Deskripsi
Reactive oxygen species (ROS) levels in gastric epithelial cells exposed to H. pylori, illustrating the oxidative stress induced by bacterial infection and its reduction by N-acetylcysteine.
Figure 2
ChartSource Paper
N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway Activation Induced by Helicobacter pylori Infection.Cite This Figure
![Figure 2: Reactive oxygen species (ROS) levels in gastric epithelial cells exposed to H. pylori, illustrating the oxidative stress induced by bacterial infection and its reduction by N-acetylcysteine.]() > Source: Chuan Xie et al. "N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway ." *Oxidative medicine and cellular longevity*, 2018. PMID: [29854076](https://pubmed.ncbi.nlm.nih.gov/29854076/)
<figure> <img src="" alt="Reactive oxygen species (ROS) levels in gastric epithelial cells exposed to H. pylori, illustrating the oxidative stress induced by bacterial infection and its reduction by N-acetylcysteine." /> <figcaption>Figure 2. Reactive oxygen species (ROS) levels in gastric epithelial cells exposed to H. pylori, illustrating the oxidative stress induced by bacterial infection and its reduction by N-acetylcysteine.<br> Source: Chuan Xie et al. "N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway ." <em>Oxidative medicine and cellular longevity</em>, 2018. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/29854076/">29854076</a></figcaption> </figure>