Description

Protein expression analysis of oxidative stress and cell signaling markers in H. pylori-infected gastric cells treated with varying concentrations of N-acetylcysteine.

Figure 9

Chart

Source Paper

N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway Activation Induced by Helicobacter pylori Infection.

Oxidative medicine and cellular longevity (2018)

PMID: 29854076

DOI: 10.1155/2018/1874985

Cite This Figure

![Figure 9: Protein expression analysis of oxidative stress and cell signaling markers in H. pylori-infected gastric cells treated with varying concentrations of N-acetylcysteine.]()

> Source: Chuan Xie et al. "N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway ." *Oxidative medicine and cellular longevity*, 2018. PMID: [29854076](https://pubmed.ncbi.nlm.nih.gov/29854076/)

<figure>
  <img src="" alt="Protein expression analysis of oxidative stress and cell signaling markers in H. pylori-infected gastric cells treated with varying concentrations of N-acetylcysteine." />
  <figcaption>Figure 9. Protein expression analysis of oxidative stress and cell signaling markers in H. pylori-infected gastric cells treated with varying concentrations of N-acetylcysteine.<br>  Source: Chuan Xie et al. "N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway ." <em>Oxidative medicine and cellular longevity</em>, 2018. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/29854076/">29854076</a></figcaption>
</figure>

Preuves associées

N-Acetyl Cysteine (NAC) pour Helicobacter pylori Infection

Note C · 3 études