Beschreibung
DNA damage assessment in H. pylori-infected gastric cells, measured by comet assay or gamma-H2AX staining, showing the protective effect of N-acetylcysteine pretreatment.
Figure 4
ChartSource Paper
N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway Activation Induced by Helicobacter pylori Infection.Cite This Figure
![Figure 4: DNA damage assessment in H. pylori-infected gastric cells, measured by comet assay or gamma-H2AX staining, showing the protective effect of N-acetylcysteine pretreatment.]() > Source: Chuan Xie et al. "N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway ." *Oxidative medicine and cellular longevity*, 2018. PMID: [29854076](https://pubmed.ncbi.nlm.nih.gov/29854076/)
<figure> <img src="" alt="DNA damage assessment in H. pylori-infected gastric cells, measured by comet assay or gamma-H2AX staining, showing the protective effect of N-acetylcysteine pretreatment." /> <figcaption>Figure 4. DNA damage assessment in H. pylori-infected gastric cells, measured by comet assay or gamma-H2AX staining, showing the protective effect of N-acetylcysteine pretreatment.<br> Source: Chuan Xie et al. "N-Acetylcysteine Reduces ROS-Mediated Oxidative DNA Damage and PI3K/Akt Pathway ." <em>Oxidative medicine and cellular longevity</em>, 2018. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/29854076/">29854076</a></figcaption> </figure>