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Germ-free and conventionalized mouse experiments delineate the requirement for gut microbiota in triggering spontaneous intestinal inflammation when epithelial mitochondrial function is impaired.
Figure 6
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Interplay of gut microbiota and host epithelial mitochondrial dysfunction is necessary for the development of spontaneous intestinal inflammation in mice.Cite This Figure
![Figure 6: Germ-free and conventionalized mouse experiments delineate the requirement for gut microbiota in triggering spontaneous intestinal inflammation when epithelial mitochondrial function is impaired.]() > Source: Kibrom M Alula et al. "Interplay of gut microbiota and host epithelial mitochondrial dysfunction is nec." *Microbiome*, 2023. PMID: [37978573](https://pubmed.ncbi.nlm.nih.gov/37978573/)
<figure> <img src="" alt="Germ-free and conventionalized mouse experiments delineate the requirement for gut microbiota in triggering spontaneous intestinal inflammation when epithelial mitochondrial function is impaired." /> <figcaption>Figure 6. Germ-free and conventionalized mouse experiments delineate the requirement for gut microbiota in triggering spontaneous intestinal inflammation when epithelial mitochondrial function is impaired.<br> Source: Kibrom M Alula et al. "Interplay of gut microbiota and host epithelial mitochondrial dysfunction is nec." <em>Microbiome</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/37978573/">37978573</a></figcaption> </figure>