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Figure 6

الوصف

Colon length measurements and macroscopic damage scoring in the colitis model, providing gross anatomical evidence of disease severity and NAC treatment response.

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Liver histopathology from colitis-induced mice with and without oral NAC supplementation, evaluating extraintestinal hepatic manifestations including steatosis and inflammatory infiltration.

Figure 2

Liver histopathology from colitis-induced mice with and without oral NAC supplementation, evaluating extraintestinal hepatic manifestations including steatosis and inflammatory infiltration.

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Kidney tissue histology showing renal changes associated with induced colitis, and the effects of N-acetylcysteine on renal inflammation and oxidative damage markers.

Figure 3

Kidney tissue histology showing renal changes associated with induced colitis, and the effects of N-acetylcysteine on renal inflammation and oxidative damage markers.

micrograph
Oxidative stress biomarkers measured in colon, liver, and kidney tissues, comparing malondialdehyde (MDA) and glutathione (GSH) levels across treatment groups.

Figure 4

Oxidative stress biomarkers measured in colon, liver, and kidney tissues, comparing malondialdehyde (MDA) and glutathione (GSH) levels across treatment groups.

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Inflammatory cytokine levels (such as TNF-alpha, IL-6, or IL-1beta) in colonic and extraintestinal tissues, assessing whether NAC modulates systemic inflammation originating from the gut.

Figure 5

Inflammatory cytokine levels (such as TNF-alpha, IL-6, or IL-1beta) in colonic and extraintestinal tissues, assessing whether NAC modulates systemic inflammation originating from the gut.

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Summary of controversial findings regarding NAC's differential effects across organs, with protective trends observed in some tissues but potential adverse signals in others.

Figure 7

Summary of controversial findings regarding NAC's differential effects across organs, with protective trends observed in some tissues but potential adverse signals in others.

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Figure 6

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Source Paper

Extraintestinal Manifestations in Induced Colitis: Controversial Effects of N-Acetylcysteine on Colon, Liver, and Kidney.

Oxidative medicine and cellular longevity (2023)

PMID: 37577725

DOI: 10.1155/2023/8811463

Cite This Figure

![Figure 6: Colon length measurements and macroscopic damage scoring in the colitis model, providing gross anatomical evidence of disease severity and NAC treatment response.](https://pdfs.citedhealth.com/figures/37577725/202.png)

> Source: Amylly Sanuelly da Paz Martins et al. "Extraintestinal Manifestations in Induced Colitis: Controversial Effects of N-Ac." *Oxidative medicine and cellular longevity*, 2023. PMID: [37577725](https://pubmed.ncbi.nlm.nih.gov/37577725/)
<figure>
  <img src="https://pdfs.citedhealth.com/figures/37577725/202.png" alt="Colon length measurements and macroscopic damage scoring in the colitis model, providing gross anatomical evidence of disease severity and NAC treatment response." />
  <figcaption>Figure 6. Colon length measurements and macroscopic damage scoring in the colitis model, providing gross anatomical evidence of disease severity and NAC treatment response.<br>  Source: Amylly Sanuelly da Paz Martins et al. "Extraintestinal Manifestations in Induced Colitis: Controversial Effects of N-Ac." <em>Oxidative medicine and cellular longevity</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/37577725/">37577725</a></figcaption>
</figure>

أدلة ذات صلة

D
N-Acetyl Cysteine (NAC) لـ Inflammatory Bowel Disease (IBD) — General

التقييم D · 1 دراسات